Essential nucleotide in the biosynthesis of phospholipids and glycolipids in cell membranes, such as neurons.

Provides polyphenols and quinones.

Precursor of the neurotransmitter Serotonin, essential for mood regulation, behavior, and anxiety.

Contributes to the normal functioning of the nervous system and normal psychological function.

Contribute to cellular protection against oxidative damage.

Aporta Astaxantina y Omega 3.

1 tablet per day.

• Up to 10 kg: 1 tablet per day.
• 10 to 20 kg: 2 tablets per day.
• 20 to 30 kg: 3 tablets per day.
• 30 to 40 kg: 4 tablets per day.
• Over 40 kg: 5 tablets per day.

Oral route

If preferred, mix with the usual food.

As it is a dietary supplement, it can be administered continuously.

Cognitive Dysfunction Syndrome (CDS) in cats and dogs is a clinical condition that affects older pets, characterized by a series of neurological disorders that negatively impact their cognitive functions, changes in behavior, social interactions, and quality of life.

Clinical signs include:

• Changes in interaction with owners or other animals.
• Disorientation and confusion, anxiety, and restlessness.
• Sleep-wake cycle disorders, changes in behavior patterns.
• Alterations in daily routines.
• Changes in motor function.

CDS has become an increasingly important area of research and veterinary care. Early diagnosis and appropriate intervention are key to addressing this syndrome.

CDS manifests in various ways:

• Macroscopic changes in the brain: cortical atrophy, which affects areas of the brain related to memory and learning, as well as the hippocampus, a structure that contributes to the formation of new memories.
• Neuronal changes: such as the loss of nerve cells in key brain areas.
• Oxidative damage and inflammation: leading to the deterioration of proteins, lipids, and nucleotides in the brain, eventually resulting in neuron death.
• Accumulation of ß-amyloid protein: may contribute to neuronal loss and brain atrophy.

CDS can also result from cerebrovascular pathologies: cerebral vascular changes, gliosis, and ventricular dilation, leading to cerebral atrophy, ventricular enlargement, and lesions in the medial temporal lobes.